A couple weeks ago you heard me tell you about a woman with
endometrial cancer who had worsening pain and bleeding and pain for a
period of six months.
She had told me that she'd stopped bleeding and had no pain for a
week.
Well, she's been on my recipe for 6 weeks now and her pain and
bleeding have been gone for three weeks. After six months of heavy
bleeding and pain, she is thrilled with her results so far.
She has received no medical treatments for her condition after
declining a hysterectomy.
The recipe consists of one freshly grated habaneros, and 2 cloves
freshly grated garlic put on sprouted bread, such as Ezekiel bread.
The peppers and garlc are smothered in butter to help quell the heat.
I'd eat this once a day. Some eat it twice a day. If my health was bad
or I had a lot of cancer, I'd go more slowly.
If I had weight loss or fluid retention from my cancer, I'd also take
6,000 mgs of evening primrose oil (EPO), in divided doses.
With no weight loss or fluid retention, I'd take a tablespoon or two
of emulsified cod liver oil each day. and not the EPO.
Take only one oil, not both, based on the one I needed at the time.
Cancer blood tests while on this diet will show high readings because
the cancer gets lysed out through the blood. If I'm feeling better and/
or observing my tumors getting smaller, I would celebrate high blood
antigen test results
Showing posts with label health. Show all posts
Showing posts with label health. Show all posts
Monday, June 1, 2009
This time my CANCER recipe only helped with her arthritis...
Jeannette wrote to me about two weeks ago. She had cancer way back in
1976, and apparently cured it with Laetrile, etc. She gave no
indication of having cancer now. She apparently decided to try it for
two weeks as a preventative.
Tonight she wrote me again. Here is what she wrote:
"I finished two weeks of the habanero, garlic, organic butter on
Ezekial 4:9 bread. On the 7th day I switched from Oil of Evening
Primrose to the emulsified cod liver oil (Twinlab) mint flavor. The
emulsified cod liver oil has relieved the chronic debilitating pain I
have had for several years in my right hip socket due to arthritis. I
am still taking Glucosamine Chondroitin & MSM tablets to restore my
joints. I wonder if that is even necessary now? I bought my second
bottle of emulsified CLO today (orange flavor this time).
"Thank you for pointing me to emulsified cod liver oil! My sister is
having the same problem and she is considering a hip replacement. As
much as I have sufferd with my hip, I have refused to consider a hip
replacement."
"I am so grateful.
-Jeannette"
It's interesting to note that she only saw improvement when she
changed the oil that she was using. I hope it continues to relieve her
pain.
1976, and apparently cured it with Laetrile, etc. She gave no
indication of having cancer now. She apparently decided to try it for
two weeks as a preventative.
Tonight she wrote me again. Here is what she wrote:
"I finished two weeks of the habanero, garlic, organic butter on
Ezekial 4:9 bread. On the 7th day I switched from Oil of Evening
Primrose to the emulsified cod liver oil (Twinlab) mint flavor. The
emulsified cod liver oil has relieved the chronic debilitating pain I
have had for several years in my right hip socket due to arthritis. I
am still taking Glucosamine Chondroitin & MSM tablets to restore my
joints. I wonder if that is even necessary now? I bought my second
bottle of emulsified CLO today (orange flavor this time).
"Thank you for pointing me to emulsified cod liver oil! My sister is
having the same problem and she is considering a hip replacement. As
much as I have sufferd with my hip, I have refused to consider a hip
replacement."
"I am so grateful.
-Jeannette"
It's interesting to note that she only saw improvement when she
changed the oil that she was using. I hope it continues to relieve her
pain.
This time my CANCER recipe only helped with her arthritis...
Jeannette wrote to me about two weeks ago. She had cancer way back in
1976, and apparently cured it with Laetrile, etc. She gave no
indication of having cancer now. She apparently decided to try it for
two weeks as a preventative.
Tonight she wrote me again. Here is what she wrote:
"I finished two weeks of the habanero, garlic, organic butter on
Ezekial 4:9 bread. On the 7th day I switched from Oil of Evening
Primrose to the emulsified cod liver oil (Twinlab) mint flavor. The
emulsified cod liver oil has relieved the chronic debilitating pain I
have had for several years in my right hip socket due to arthritis. I
am still taking Glucosamine Chondroitin & MSM tablets to restore my
joints. I wonder if that is even necessary now? I bought my second
bottle of emulsified CLO today (orange flavor this time).
"Thank you for pointing me to emulsified cod liver oil! My sister is
having the same problem and she is considering a hip replacement. As
much as I have sufferd with my hip, I have refused to consider a hip
replacement."
"I am so grateful.
-Jeannette"
It's interesting to note that she only saw improvement when she
changed the oil that she was using. I hope it continues to relieve her
pain.
1976, and apparently cured it with Laetrile, etc. She gave no
indication of having cancer now. She apparently decided to try it for
two weeks as a preventative.
Tonight she wrote me again. Here is what she wrote:
"I finished two weeks of the habanero, garlic, organic butter on
Ezekial 4:9 bread. On the 7th day I switched from Oil of Evening
Primrose to the emulsified cod liver oil (Twinlab) mint flavor. The
emulsified cod liver oil has relieved the chronic debilitating pain I
have had for several years in my right hip socket due to arthritis. I
am still taking Glucosamine Chondroitin & MSM tablets to restore my
joints. I wonder if that is even necessary now? I bought my second
bottle of emulsified CLO today (orange flavor this time).
"Thank you for pointing me to emulsified cod liver oil! My sister is
having the same problem and she is considering a hip replacement. As
much as I have sufferd with my hip, I have refused to consider a hip
replacement."
"I am so grateful.
-Jeannette"
It's interesting to note that she only saw improvement when she
changed the oil that she was using. I hope it continues to relieve her
pain.
Anti-inflammatory Soy Lecithin
Research Article
Soy lecithin supplementation alters macrophage phagocytosis and
lymphocyte response to concanavalin A: a study in alloxan-induced
diabetic rats
Dalva T. S. Z. Miranda 1, Vanessa G. Batista 1, Fernanda C. C. Grando
1, Fernanda M. Paula 1, Caroline A. Felício 1, Gabriella F. S. Rubbo
1, Luiz C. Fernandes 1, Rui Curi 2, Anita Nishiyama 1 *
1Department of Physiology, Universidade Federal do Paraná, Curitiba,
PR, Brazil
2Department of Physiology and Biophysics, Universidade de São Paulo,
SP, Brazil
email: Anita Nishiyama (anita.ni...@ufpr.br)
*Correspondence to Anita Nishiyama, Department of Physiology,
Universidade Federal do Paraná, Curitiba, PR, Brazil.
Abstract
Dietary soy lecithin supplementation decreases hyperlipidemia and
influences lipid metabolism.
Although this product is used by diabetic patients, there are no data
about the effect of soy lecithin supplementation on the immune
system.
The addition of phosphatidylcholine, the main component of lecithin,
to a culture of lymphocytes has been reported to alter their
function.
If phosphatidylcholine changes lymphocyte functions in vitro as
previously shown, then it could also affect immune cells in vivo.
In the present study, the effect of dietary soy lecithin on macrophage
phagocytic capacity and on lymphocyte number in response to
concanavalin A (ConA) stimulation was investigated in non-diabetic and
alloxan-induced diabetic rats. Supplementation was carried out daily
with 2 g kg-1 b.w. lecithin during 7 days.
After that, blood was drawn from fasting rats and peritoneal
macrophages and mesenteric lymph node lymphocytes were collected to
determine the phospholipid content.
Plasma triacylglycerol (TAG), total and HDL cholesterol and glucose
levels were also determined. Lymphocytes were stimulated by ConA. The
MTT (3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide) dye
reduction method and flow cytometry were employed to evaluate
lymphocyte metabolism and cell number, respectively.
Soy lecithin supplementation significantly increased both macrophage
phagocytic capacity (+29%) in non-diabetic rats and the lymphocyte
number in diabetic rats (+92%).
It is unlikely that plasma lipid levels indirectly affect immune
cells, since plasma cholesterol, TAG, or phospholipid content was not
modified by lecithin supplementation.
In conclusion, lymphocyte and macrophage function were altered by
lecithin supplementation, indicating an immunomodulatory effect of
phosphatidylcholine.
Funded by:
CNPQ; Grant Number: 473412/2003-9
Keywords
dietary supplements • phosphatidylcholines • macrophages • lymphocytes
• diabetes mellitus
Received: 4 June 2008; Revised: 26 August 2008; Accepted: 3 September
2008
Digital Object Identifier (DOI)
10.1002/cbf.1517 About DOI
Copyright © 2008 John Wiley & Sons, Ltd.
Soy lecithin supplementation alters macrophage phagocytosis and
lymphocyte response to concanavalin A: a study in alloxan-induced
diabetic rats
Dalva T. S. Z. Miranda 1, Vanessa G. Batista 1, Fernanda C. C. Grando
1, Fernanda M. Paula 1, Caroline A. Felício 1, Gabriella F. S. Rubbo
1, Luiz C. Fernandes 1, Rui Curi 2, Anita Nishiyama 1 *
1Department of Physiology, Universidade Federal do Paraná, Curitiba,
PR, Brazil
2Department of Physiology and Biophysics, Universidade de São Paulo,
SP, Brazil
email: Anita Nishiyama (anita.ni...@ufpr.br)
*Correspondence to Anita Nishiyama, Department of Physiology,
Universidade Federal do Paraná, Curitiba, PR, Brazil.
Abstract
Dietary soy lecithin supplementation decreases hyperlipidemia and
influences lipid metabolism.
Although this product is used by diabetic patients, there are no data
about the effect of soy lecithin supplementation on the immune
system.
The addition of phosphatidylcholine, the main component of lecithin,
to a culture of lymphocytes has been reported to alter their
function.
If phosphatidylcholine changes lymphocyte functions in vitro as
previously shown, then it could also affect immune cells in vivo.
In the present study, the effect of dietary soy lecithin on macrophage
phagocytic capacity and on lymphocyte number in response to
concanavalin A (ConA) stimulation was investigated in non-diabetic and
alloxan-induced diabetic rats. Supplementation was carried out daily
with 2 g kg-1 b.w. lecithin during 7 days.
After that, blood was drawn from fasting rats and peritoneal
macrophages and mesenteric lymph node lymphocytes were collected to
determine the phospholipid content.
Plasma triacylglycerol (TAG), total and HDL cholesterol and glucose
levels were also determined. Lymphocytes were stimulated by ConA. The
MTT (3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide) dye
reduction method and flow cytometry were employed to evaluate
lymphocyte metabolism and cell number, respectively.
Soy lecithin supplementation significantly increased both macrophage
phagocytic capacity (+29%) in non-diabetic rats and the lymphocyte
number in diabetic rats (+92%).
It is unlikely that plasma lipid levels indirectly affect immune
cells, since plasma cholesterol, TAG, or phospholipid content was not
modified by lecithin supplementation.
In conclusion, lymphocyte and macrophage function were altered by
lecithin supplementation, indicating an immunomodulatory effect of
phosphatidylcholine.
Funded by:
CNPQ; Grant Number: 473412/2003-9
Keywords
dietary supplements • phosphatidylcholines • macrophages • lymphocytes
• diabetes mellitus
Received: 4 June 2008; Revised: 26 August 2008; Accepted: 3 September
2008
Digital Object Identifier (DOI)
10.1002/cbf.1517 About DOI
Copyright © 2008 John Wiley & Sons, Ltd.
AUTISM: What Happens To Victims After Parents Die? Dark Days Loom ...
"Most of these cognitively impaired citizens don't vote. Most of them
can't live alone or work in public places. Many can't even take public
transportation by themselves"
"The number of autistic children expected to need extensive adult
services by 2023 -- more than 380,000 people -- is roughly equal to
the population of Minneapolis."
----------------
"Still Overlooking Autistic Adults"
Op-Ed
By Linda H. Davis
Saturday, April 4, 2009; A15
Question: What coming social expenditure will cost more than a third
of this year's budget for the Department of Health and Human Services
and be larger than the entire current budget of the Energy
Department?
Answer: The bill for the tide of autistic children entering adulthood
over the next 15 years, an estimated $27 billion annually in current,
non-inflation-adjusted dollars by the end of that period. The number
of autistic children expected to need extensive adult services by 2023
-- more than 380,000 people -- is roughly equal to the population of
Minneapolis. If a town were created to house this group of people and
their caregivers -- for you can't separate the two -- it would exceed
the population of all but six U.S. cities. If they formed a state, it
would have four electoral votes.
But most of these cognitively impaired citizens don't vote. Most of
them can't live alone or work in public places. Many can't even take
public transportation by themselves.
Yet as World Autism Awareness Day passed this week, with the wrecking
ball swinging at all levels of social services in this devastated
economy, the challenges of adult autism continue to be overlooked.
Many news reports focus on whether vaccines cause autism, the need for
a cure or the education of autistic children. Autistic adults are
relegated to the sidelines. Even the Obama administration, which has
pledged better care for disabled Americans, including those with
autism, has not been specific enough about its plans for those who
will probably never be able to live independently or be part of the
traditional workforce. "Improving life-long services for people with
ASD," as autism spectrum disorder is known, is a worryingly broad,
detail-free promise in the White House agenda published online.
I understand that no one wants to look at a child and imagine the
clunky, in-your-face adult he or she will become or think about the
stares he or she will induce. When I look at my pudgy 22-year-old son,
Randy, still sweet-faced but so obviously disabled, I cannot locate
the blond cherub he used to be, gripping his stuffed brown bear. While
writing this, I listened to Randy getting into the refrigerator (he's
home again from his supervised job -- two mornings a week -- because
of another problem with the overwhelmed human services provider funded
by the Commonwealth of Massachusetts), and wonder who will love or at
least protect him when he ends up in a group home run by an underpaid,
overworked staff.
Randy's father and I are keeping him at home as long as possible, even
as I'm battling an incurable cancer. The inadequate state services and
perilously thin funding have seriously hampered our ability to work
while caring for our son. I feel as though we're playing Russian
roulette with Randy's future, yet I cling to my gentle son, unwilling
to entrust him to a system that grows more fragile than I do.
Randy is just one of hundreds of thousands of autistic adults to whom
society pays frighteningly little attention. The price of their care
will affect all Americans, not only those who have autistic children.
Along with housing, day programs, transportation to those programs or
jobs, and higher-than-average medical costs, adults with autism
require steady supervision and support. Consider: A well-behaved,
relatively high-functioning person such as my son could manage in an
environment that has a ratio of three clients per staff member. But
many autistic people require a one-to-one ratio. This is a serious
hurdle, not least because of the high turnover rate among those who
provide direct care, which stems in part from their low wages. Not
everyone is temperamentally suited to this work. People with autism
present myriad challenges: They can sometimes be violent, sometimes
are self-abusive, suffer psychological meltdowns, or behave in many
socially unacceptable ways, to say the least. Women, traditionally
cast in the caregiver role, are at risk of greater physical harm when
caring for autistic adults than for children. At expected rates, we
will need to find an additional million caregivers, people who must
have the right personal qualities to work with autistic individuals
but who are willing and able to work for low wages. This is no small
challenge. We not only must train people but also show that we value
this work by paying them better.
In 15 years, the cost of care just for the autistic children entering
adulthood over that time will be about equal to the current state
budget of Tennessee. Meanwhile, services are dangerously strained, and
the influx of autistic adults is underway. This country urgently needs
to focus on adult autism, new models of care and new sources of
funding. Before the looming tidal wave delivers another crushing blow
to our economy, we should have a national discussion. It should begin
today.
[Linda H. Davis is the author, most recently, of "Charles Addams: A
Cartoonist's Life" and is president of the nonprofit SAGE Crossing
Foundation, formed to create a farmstead for autistic adults. Her
husband, Chuck Yanikoski, who is treasurer of SAGE, contributed to
this piece.]
can't live alone or work in public places. Many can't even take public
transportation by themselves"
"The number of autistic children expected to need extensive adult
services by 2023 -- more than 380,000 people -- is roughly equal to
the population of Minneapolis."
----------------
"Still Overlooking Autistic Adults"
Op-Ed
By Linda H. Davis
Saturday, April 4, 2009; A15
Question: What coming social expenditure will cost more than a third
of this year's budget for the Department of Health and Human Services
and be larger than the entire current budget of the Energy
Department?
Answer: The bill for the tide of autistic children entering adulthood
over the next 15 years, an estimated $27 billion annually in current,
non-inflation-adjusted dollars by the end of that period. The number
of autistic children expected to need extensive adult services by 2023
-- more than 380,000 people -- is roughly equal to the population of
Minneapolis. If a town were created to house this group of people and
their caregivers -- for you can't separate the two -- it would exceed
the population of all but six U.S. cities. If they formed a state, it
would have four electoral votes.
But most of these cognitively impaired citizens don't vote. Most of
them can't live alone or work in public places. Many can't even take
public transportation by themselves.
Yet as World Autism Awareness Day passed this week, with the wrecking
ball swinging at all levels of social services in this devastated
economy, the challenges of adult autism continue to be overlooked.
Many news reports focus on whether vaccines cause autism, the need for
a cure or the education of autistic children. Autistic adults are
relegated to the sidelines. Even the Obama administration, which has
pledged better care for disabled Americans, including those with
autism, has not been specific enough about its plans for those who
will probably never be able to live independently or be part of the
traditional workforce. "Improving life-long services for people with
ASD," as autism spectrum disorder is known, is a worryingly broad,
detail-free promise in the White House agenda published online.
I understand that no one wants to look at a child and imagine the
clunky, in-your-face adult he or she will become or think about the
stares he or she will induce. When I look at my pudgy 22-year-old son,
Randy, still sweet-faced but so obviously disabled, I cannot locate
the blond cherub he used to be, gripping his stuffed brown bear. While
writing this, I listened to Randy getting into the refrigerator (he's
home again from his supervised job -- two mornings a week -- because
of another problem with the overwhelmed human services provider funded
by the Commonwealth of Massachusetts), and wonder who will love or at
least protect him when he ends up in a group home run by an underpaid,
overworked staff.
Randy's father and I are keeping him at home as long as possible, even
as I'm battling an incurable cancer. The inadequate state services and
perilously thin funding have seriously hampered our ability to work
while caring for our son. I feel as though we're playing Russian
roulette with Randy's future, yet I cling to my gentle son, unwilling
to entrust him to a system that grows more fragile than I do.
Randy is just one of hundreds of thousands of autistic adults to whom
society pays frighteningly little attention. The price of their care
will affect all Americans, not only those who have autistic children.
Along with housing, day programs, transportation to those programs or
jobs, and higher-than-average medical costs, adults with autism
require steady supervision and support. Consider: A well-behaved,
relatively high-functioning person such as my son could manage in an
environment that has a ratio of three clients per staff member. But
many autistic people require a one-to-one ratio. This is a serious
hurdle, not least because of the high turnover rate among those who
provide direct care, which stems in part from their low wages. Not
everyone is temperamentally suited to this work. People with autism
present myriad challenges: They can sometimes be violent, sometimes
are self-abusive, suffer psychological meltdowns, or behave in many
socially unacceptable ways, to say the least. Women, traditionally
cast in the caregiver role, are at risk of greater physical harm when
caring for autistic adults than for children. At expected rates, we
will need to find an additional million caregivers, people who must
have the right personal qualities to work with autistic individuals
but who are willing and able to work for low wages. This is no small
challenge. We not only must train people but also show that we value
this work by paying them better.
In 15 years, the cost of care just for the autistic children entering
adulthood over that time will be about equal to the current state
budget of Tennessee. Meanwhile, services are dangerously strained, and
the influx of autistic adults is underway. This country urgently needs
to focus on adult autism, new models of care and new sources of
funding. Before the looming tidal wave delivers another crushing blow
to our economy, we should have a national discussion. It should begin
today.
[Linda H. Davis is the author, most recently, of "Charles Addams: A
Cartoonist's Life" and is president of the nonprofit SAGE Crossing
Foundation, formed to create a farmstead for autistic adults. Her
husband, Chuck Yanikoski, who is treasurer of SAGE, contributed to
this piece.]
DHA Found to Shrink Tumors, Aid Use of Chemotherapy Drug
DHA, an omega-3 fatty acid found in fish oils, has been shown to
reduce the size of tumors and enhance the positive effects of the
chemotherapy drug cisplatin, while limiting its harmful side effects.
These experiments, described in the journal "Cell Division," provide
some support for the many health benefits often suggested for omega-3
acids.
Professor A. M. El-Mowafy and his team from Mansoura University
(Egypt) studied DHA’s effects on solid tumors growing in mice, and
investigated how this fatty acid interacts with cisplatin, a
chemotherapy drug that is known to cause kidney damage. El-Mowafy
said, “DHA elicited prominent chemopreventive effects on its own, and
appreciably augmented those of cisplatin as well." He went further and
indicated that DHA can obliterate certain cisplatin-induced toxicity
and damage to kidney tissue.
DHA is an omega-3 fatty acid that is commonly found in cold-water fish
oil, and some vegetable oils. It is a major component of brain gray
matter and of the retina in most mammalian species and is considered
essential for normal neurological and cellular developments. This
study found that, at the molecular level, DHA acts by reducing white
blood cell accumulation, inflammation, and oxidative stress – all
processes that have been linked with tumor growth.
El-Mowafy and his colleagues have called for greater deployment of
omega-3 in the fight against cancer.
Dave
Full text article above extracted from http://shamvswham.blogspot.com/
reduce the size of tumors and enhance the positive effects of the
chemotherapy drug cisplatin, while limiting its harmful side effects.
These experiments, described in the journal "Cell Division," provide
some support for the many health benefits often suggested for omega-3
acids.
Professor A. M. El-Mowafy and his team from Mansoura University
(Egypt) studied DHA’s effects on solid tumors growing in mice, and
investigated how this fatty acid interacts with cisplatin, a
chemotherapy drug that is known to cause kidney damage. El-Mowafy
said, “DHA elicited prominent chemopreventive effects on its own, and
appreciably augmented those of cisplatin as well." He went further and
indicated that DHA can obliterate certain cisplatin-induced toxicity
and damage to kidney tissue.
DHA is an omega-3 fatty acid that is commonly found in cold-water fish
oil, and some vegetable oils. It is a major component of brain gray
matter and of the retina in most mammalian species and is considered
essential for normal neurological and cellular developments. This
study found that, at the molecular level, DHA acts by reducing white
blood cell accumulation, inflammation, and oxidative stress – all
processes that have been linked with tumor growth.
El-Mowafy and his colleagues have called for greater deployment of
omega-3 in the fight against cancer.
Dave
Full text article above extracted from http://shamvswham.blogspot.com/
BLOODLETTING IN PNEUMONIA
John Haddon
BLOODLETTING IN PNEUMONIA.
Sir, Dr. Balms's communication in the Journal of June 5th, p. 970,
telling how he treats some cases of pneumonia, deserves to be
noticed.
It was the late Professor John Jughes Bennet who first advocated the
expectant treatment of pneumonia, and his book on the subject was
translated into many languages.
I acted as his resident in the clinical wards of the Edinburgh Royal
Infirmary, and , by his directions, I occasionally bled patients.
Bennet would have bled the cases Dr. Balm describes , and if the idea
is original on his part , he deserves credit.
Talking about his letter with a medical friend , he told me that he
had a patient whose nose bled profusely; he failed to stop it, and the
late Dr. Joseph Bell was consulted he told my friend that his father
would , in such a case , have opened a vein, and that was what Dr.
Joseph Bell did.
He did not take more than two tablespoonsfuls of blood , and there was
no more bleeding from the nose.
Our grandfathers used to be bled every spring, and I have heard
Professor Bennet tell of the row of patients waiting to be bled.
Lanquid and lazy before being bled, they felt as if their youth were
renewed by the bleeding.
I heard Sir T. Clifford Allbutt tell of having had a patient suffering
from a pulse of very high tension, who was kept for a year by one
venesection; in these days of so-called sudden death from heart
failure or apoplexy , which I look upon as an opprobrium to the
physician, it would be well to resort to an annual bleeding aagain,
unless the profession can be converted to the views set forth in my
book which proves that food is the chief cause of disease, and
restricts the quantity , as well as changes the quality , of the
patients food.
In what has been called "idiopathic anasarca" we have a neurosis , due
to what the ancient physicians called plethora, which was just too
much blood, and which venesecton removed.
Such anasarca is almost universal after middle life, and I have found
it in some quite young , proving that even the youngest may be injured
by the food they eat.
Such facts ought to encourage the study of dietetics which the General
Medical Council would do well to make a compulsory subject of
examination for every licence to practice medicine. -- I am, etc.,
Hawick June 7th, John Haddon, M.D.
Who loves ya.
Tom
BLOODLETTING IN PNEUMONIA.
Sir, Dr. Balms's communication in the Journal of June 5th, p. 970,
telling how he treats some cases of pneumonia, deserves to be
noticed.
It was the late Professor John Jughes Bennet who first advocated the
expectant treatment of pneumonia, and his book on the subject was
translated into many languages.
I acted as his resident in the clinical wards of the Edinburgh Royal
Infirmary, and , by his directions, I occasionally bled patients.
Bennet would have bled the cases Dr. Balm describes , and if the idea
is original on his part , he deserves credit.
Talking about his letter with a medical friend , he told me that he
had a patient whose nose bled profusely; he failed to stop it, and the
late Dr. Joseph Bell was consulted he told my friend that his father
would , in such a case , have opened a vein, and that was what Dr.
Joseph Bell did.
He did not take more than two tablespoonsfuls of blood , and there was
no more bleeding from the nose.
Our grandfathers used to be bled every spring, and I have heard
Professor Bennet tell of the row of patients waiting to be bled.
Lanquid and lazy before being bled, they felt as if their youth were
renewed by the bleeding.
I heard Sir T. Clifford Allbutt tell of having had a patient suffering
from a pulse of very high tension, who was kept for a year by one
venesection; in these days of so-called sudden death from heart
failure or apoplexy , which I look upon as an opprobrium to the
physician, it would be well to resort to an annual bleeding aagain,
unless the profession can be converted to the views set forth in my
book which proves that food is the chief cause of disease, and
restricts the quantity , as well as changes the quality , of the
patients food.
In what has been called "idiopathic anasarca" we have a neurosis , due
to what the ancient physicians called plethora, which was just too
much blood, and which venesecton removed.
Such anasarca is almost universal after middle life, and I have found
it in some quite young , proving that even the youngest may be injured
by the food they eat.
Such facts ought to encourage the study of dietetics which the General
Medical Council would do well to make a compulsory subject of
examination for every licence to practice medicine. -- I am, etc.,
Hawick June 7th, John Haddon, M.D.
Who loves ya.
Tom
Acute Stroke Treatment
Efficacy of citicoline as an acute stroke treatment.
Expert Opin Pharmacother 2009 Apr; 10(5):839-46.
Clark WM
Citicoline (cytidine-5'-diphosphocholine or CDP-choline) is a
precursor essential for the synthesis of phosphatidylcholine,
one of the cell membrane components that is degraded during
cerebral ischemia to free fatty acids and free radicals.
Animal studies suggest that citicoline may protect cell membranes
by accelerating resynthesis of phospholipids and suppressing the
release of free fatty acids, stabilizing cell membranes, and reducing
free radical generation.
Numerous experimental stroke studies with citicoline have shown
improved outcome and reduced infarct size in both ischemic and
hemorrhagic stroke models.
Citicoline has been studied worldwide in both ischemic and
hemorrhagic
clinical stroke with excellent safety and possibly efficacy found in
several
trials.
A meta-analysis of four randomized US clinical citicoline trials
concluded
that treatment with oral citicoline within the first 24 h after a
moderate to
severe stroke is safe and increases the probability of complete
recovery
at 3 months.
Citicoline clinical efficacy trials are now continuing outside of the
US in
both ischemic and hemorrhagic stroke.
A citicoline supplement is now available from several sources on the
internet.
Expert opinion on pharmacotherapy [Expert Opin Pharmacother]
----------
"The mechanism may be that soybean lecithin lowers
the decrease of brain phospholipid content in brain ischemia"
Curative effect of soybean lecithin on cerebral infarction
Shi F, Zhou J, Meng D
Zhonghua Yi Xue Za Zhi. 2001 Nov 10; 81(21): 1301-3
OBJECTIVE:
To investigate the clinical curative effect of soybean lecithin on
cerebral infarction.
METHODS:
542 patients with cerebral infarction within 48 h after the onset
with the nervous fuction defect scores of 31-35 were divided into
3 groups: basic treatment group, 60 cases, with conventional
treatment; citicoline group, 122 patients, with conventional
treatment and citicoline; and soybean lecithin group, 360 patients,
with conventional treatment and soybean lecithin 10 g tid.
For all patients, treatment began sometime within 48 hours after
the onset.
The treatment course lasted 28 days.
RESULTS:
When the course was over, the infarct volumes in basic group
citicoline group, and soybean lecithin group, were 7.6 cm3 +/- 2.9
cm3,
7.3 cm3 +/- 3.1 cm3, and 6.4 cm3 +/- 2.7 cm3 respectively
(F = 7.371, P = 0.0007).
The basic group and citicoline group being compared with the soybean
group by Dunnett test, t = 4.387 and 3.969 respectively, P < 0.01.
The nervous function defect integral in the 3 groups decreased 14.2
+/-
10.93, 15.0 +/- 9.0, and 18.5 +/- 10.9 respectively.
Two-way analysis of variance of drug kind and beginning time of
treatment
showed the value of F in drugs as 6.250, P = 0.0021, and value of F in
times
as 0.9417, P = 0.4201. In the order of death,deterioration,
nonimprovement,
improvement, significant improvement, and recovery, the Ridit values
for the
comprehensive curative effect in the 3 groups were 0.4003, 0.4118,
and
0.54 5 respectively; chi2 = 27.89, P < 0.001.
CONCLUSION:
Soybean lecithin is effective in treatment of acute cerebral
infarction.
The mechanism may be that soybean lecithin lowers the decrease of
brain
phospholipid content in brain ischemia.
-----------
Who loves ya.
Tom
Expert Opin Pharmacother 2009 Apr; 10(5):839-46.
Clark WM
Citicoline (cytidine-5'-diphosphocholine or CDP-choline) is a
precursor essential for the synthesis of phosphatidylcholine,
one of the cell membrane components that is degraded during
cerebral ischemia to free fatty acids and free radicals.
Animal studies suggest that citicoline may protect cell membranes
by accelerating resynthesis of phospholipids and suppressing the
release of free fatty acids, stabilizing cell membranes, and reducing
free radical generation.
Numerous experimental stroke studies with citicoline have shown
improved outcome and reduced infarct size in both ischemic and
hemorrhagic stroke models.
Citicoline has been studied worldwide in both ischemic and
hemorrhagic
clinical stroke with excellent safety and possibly efficacy found in
several
trials.
A meta-analysis of four randomized US clinical citicoline trials
concluded
that treatment with oral citicoline within the first 24 h after a
moderate to
severe stroke is safe and increases the probability of complete
recovery
at 3 months.
Citicoline clinical efficacy trials are now continuing outside of the
US in
both ischemic and hemorrhagic stroke.
A citicoline supplement is now available from several sources on the
internet.
Expert opinion on pharmacotherapy [Expert Opin Pharmacother]
----------
"The mechanism may be that soybean lecithin lowers
the decrease of brain phospholipid content in brain ischemia"
Curative effect of soybean lecithin on cerebral infarction
Shi F, Zhou J, Meng D
Zhonghua Yi Xue Za Zhi. 2001 Nov 10; 81(21): 1301-3
OBJECTIVE:
To investigate the clinical curative effect of soybean lecithin on
cerebral infarction.
METHODS:
542 patients with cerebral infarction within 48 h after the onset
with the nervous fuction defect scores of 31-35 were divided into
3 groups: basic treatment group, 60 cases, with conventional
treatment; citicoline group, 122 patients, with conventional
treatment and citicoline; and soybean lecithin group, 360 patients,
with conventional treatment and soybean lecithin 10 g tid.
For all patients, treatment began sometime within 48 hours after
the onset.
The treatment course lasted 28 days.
RESULTS:
When the course was over, the infarct volumes in basic group
citicoline group, and soybean lecithin group, were 7.6 cm3 +/- 2.9
cm3,
7.3 cm3 +/- 3.1 cm3, and 6.4 cm3 +/- 2.7 cm3 respectively
(F = 7.371, P = 0.0007).
The basic group and citicoline group being compared with the soybean
group by Dunnett test, t = 4.387 and 3.969 respectively, P < 0.01.
The nervous function defect integral in the 3 groups decreased 14.2
+/-
10.93, 15.0 +/- 9.0, and 18.5 +/- 10.9 respectively.
Two-way analysis of variance of drug kind and beginning time of
treatment
showed the value of F in drugs as 6.250, P = 0.0021, and value of F in
times
as 0.9417, P = 0.4201. In the order of death,deterioration,
nonimprovement,
improvement, significant improvement, and recovery, the Ridit values
for the
comprehensive curative effect in the 3 groups were 0.4003, 0.4118,
and
0.54 5 respectively; chi2 = 27.89, P < 0.001.
CONCLUSION:
Soybean lecithin is effective in treatment of acute cerebral
infarction.
The mechanism may be that soybean lecithin lowers the decrease of
brain
phospholipid content in brain ischemia.
-----------
Who loves ya.
Tom
Low Vitamin A, C Intake Tied to Asthma Risk
THURSDAY, April 16 (HealthDay News) -- People who don't get enough of the antioxidant vitamins A and C in their diet may be at increased risk for asthma, British researchers say.
The pooled results of 40 studies conducted between 1980 and 2007 showed that people with asthma had a significantly lower dietary intake of vitamin A than those without the disease. The average intake among those with asthma was 182 micrograms a day, which is between a quarter and a third of recommended daily intake.
The review authors also found that people with severe asthma had a significantly lower intake of vitamin C (about half the recommended daily intake) than those with mild asthma. In addition, low circulating levels of vitamin C in the blood and lower dietary intake of foods containing vitamin C were associated with a 12 percent increased risk of asthma.
There was no association between vitamin E intake and asthma risk, but blood levels of vitamin E were much lower among people with severe asthma than in those with mild asthma. Those with severe asthma had an average vitamin E intake of 2 milligrams/day, which is 20 percent lower than the daily recommended amount, the review authors said.
These findings don't prove cause and effect, but they do challenge a study published last year that found no association between antioxidants and asthma risk, said Dr. Jo Leonardi-Bee, of the division of epidemiology and public health at the University of Nottingham in the United Kingdom, and colleagues.
"Overall, our findings from [the current] systematic review and meta-analysis indicate that low levels of vitamin C intake, and to a lesser extent vitamin A, are consistently associated with asthma risk to a degree that, if causal, would be sufficient to be clinically relevant," they concluded.
Their findings for an association between dietary antioxidants and wheezing were less consistent. The report was published in the current issue of Thorax.
More information
The U.S. National Heart, Lung, and Blood Institute has more about asthma.
-- Robert Preidt
SOURCE: BMJ Specialist Journals, news release, April 15, 2009
Last Updated: April 16, 2009
The pooled results of 40 studies conducted between 1980 and 2007 showed that people with asthma had a significantly lower dietary intake of vitamin A than those without the disease. The average intake among those with asthma was 182 micrograms a day, which is between a quarter and a third of recommended daily intake.
The review authors also found that people with severe asthma had a significantly lower intake of vitamin C (about half the recommended daily intake) than those with mild asthma. In addition, low circulating levels of vitamin C in the blood and lower dietary intake of foods containing vitamin C were associated with a 12 percent increased risk of asthma.
There was no association between vitamin E intake and asthma risk, but blood levels of vitamin E were much lower among people with severe asthma than in those with mild asthma. Those with severe asthma had an average vitamin E intake of 2 milligrams/day, which is 20 percent lower than the daily recommended amount, the review authors said.
These findings don't prove cause and effect, but they do challenge a study published last year that found no association between antioxidants and asthma risk, said Dr. Jo Leonardi-Bee, of the division of epidemiology and public health at the University of Nottingham in the United Kingdom, and colleagues.
"Overall, our findings from [the current] systematic review and meta-analysis indicate that low levels of vitamin C intake, and to a lesser extent vitamin A, are consistently associated with asthma risk to a degree that, if causal, would be sufficient to be clinically relevant," they concluded.
Their findings for an association between dietary antioxidants and wheezing were less consistent. The report was published in the current issue of Thorax.
More information
The U.S. National Heart, Lung, and Blood Institute has more about asthma.
-- Robert Preidt
SOURCE: BMJ Specialist Journals, news release, April 15, 2009
Last Updated: April 16, 2009
Pancreatic cancer therapy
Pancreatic cancer often goes undetected
Promising early results for a drug for pancreatic cancer have been reported by a team of UK and US scientists.
The drug, which targets a molecule called PKD involved in tumour growth, also seemed effective in animal tests on lung cancer, the researchers said.
The findings are especially encouraging because there are few treatments available and survival is poor.
Human trials should start within 18 months, the American Association for Cancer Research conference was told.
PKD is a family of molecules called kinases which provide a signalling function between the outside and inside of the cell.
Also involved in cell survival and the formation of new blood vessels, PKD was discovered to be potentially key target in tumours by UK researchers some years ago.
A team at Cancer Research Technology Ltd - a company owned by Cancer Research UK - then developed molecules which would inhibit the effects of PKD.
The latest results on the resulting drug, known as CRT0066101, show it inhibits the growth of pancreatic tumours in mice and works in lung cancer models.
It is thought that future studies may show the drug to be effective on a wider range of cancers.
Human trials should be starting after safety studies have been completed, they researchers said.
'Unmet need'
CRT's discovery laboratories director Dr Hamish Ryder said the team focused on pancreatic and lung cancer tumours because they are cancers with a "significant unmet medical need".
Dr Sushovan Guha, who leads the laboratory at MD Anderson Cancer Center and collaborated in the project, added he was optimistic about the drug's potential.
In addition to killing cancer cells, it is hoped the drug will stop tumours growing and spreading by blocking blood vessel growth.
"This would mean it offers a double action treatment but this needs to be proved through further work."
Sue Ballard, the founder of Pancreatic Cancer UK, said the disease caused 5% of cancer deaths but only received 1% of disease funding.
"There is a great lack of really effective treatments, surgery gives the best chance if done early but even in that situation it can recur or spread.
"This research is in the very early stages but anything that's starting to show promising results is vitally needed."
Promising early results for a drug for pancreatic cancer have been reported by a team of UK and US scientists.
The drug, which targets a molecule called PKD involved in tumour growth, also seemed effective in animal tests on lung cancer, the researchers said.
The findings are especially encouraging because there are few treatments available and survival is poor.
Human trials should start within 18 months, the American Association for Cancer Research conference was told.
PKD is a family of molecules called kinases which provide a signalling function between the outside and inside of the cell.
Also involved in cell survival and the formation of new blood vessels, PKD was discovered to be potentially key target in tumours by UK researchers some years ago.
A team at Cancer Research Technology Ltd - a company owned by Cancer Research UK - then developed molecules which would inhibit the effects of PKD.
The latest results on the resulting drug, known as CRT0066101, show it inhibits the growth of pancreatic tumours in mice and works in lung cancer models.
It is thought that future studies may show the drug to be effective on a wider range of cancers.
Human trials should be starting after safety studies have been completed, they researchers said.
'Unmet need'
CRT's discovery laboratories director Dr Hamish Ryder said the team focused on pancreatic and lung cancer tumours because they are cancers with a "significant unmet medical need".
Dr Sushovan Guha, who leads the laboratory at MD Anderson Cancer Center and collaborated in the project, added he was optimistic about the drug's potential.
In addition to killing cancer cells, it is hoped the drug will stop tumours growing and spreading by blocking blood vessel growth.
"This would mean it offers a double action treatment but this needs to be proved through further work."
Sue Ballard, the founder of Pancreatic Cancer UK, said the disease caused 5% of cancer deaths but only received 1% of disease funding.
"There is a great lack of really effective treatments, surgery gives the best chance if done early but even in that situation it can recur or spread.
"This research is in the very early stages but anything that's starting to show promising results is vitally needed."
Friday, May 1, 2009
How to Choose a Dry Skin Cream by Frank Langella
Are you frustrated with dry, flaky skin? Are you looking for a dry skin cream that can make your skin soft and smooth again? Well, you are in luck. Finding such a cream is really not difficult; there are just a few things you need to look for. Let's find out what they are.
Why does it happen
Before we talk about choosing a dry skin cream, we need to understand why does our skin get dry in the first place. Broadly, there are two causes of dry skin:
1) Environmental factors - wind, sun and even cold weather cause the skin to become dry
2) Use of soap and harsh cleansers - soaps wash out the natural oils present in our skin, which then makes our skin dry. A similar effect is brought upon our skin by using harsh cleansers. Ideally, a cleanser should only remove extra oil and the dust or pollution that has settled down on the skin. But many cleansers remove all the oils from the skin, making it deficient in oil and hence, dry.
The solution
Some basic skin care combined with a good, natural dry skin cream will work wonders in getting your skin back in shape. A balanced diet is essential for making sure your skin is nourished and gets all the nutrients to keep it healthy. Drink plenty of water - this will keep your body and skin hydrated naturally.
Top it off with a natural skin cream. Now, it's easy to rush off to your neighborhood store and buy the first cream that is packaged attractively and looks good. That, however, is not the best way to do it.
Instead, just do a little research and check the ingredients that a good skin cream should have. Most of the creams available in the stores contain harmful chemicals that do more damage than good for your skin. What is needed is a cream with natural ingredients like Active Manuka Honey. This is a special honey from New Zealand which helps keep the skin hydrated.
Babassu wax is another natural ingredient which softens and soothes the skin. It is made from Babassu oil - sourced from the Babassu palm found in Brazil. It creates a thin, invisible layer on the skin which keeps the dust out and retains the moisture inside.
Do yourself a favor and find out a good natural dry skin cream containing these natural ingredients. You can have a baby-soft smooth skin back in no time!
About the Author
For information on hand lotion to lighten age spots, visit my website today.
Frank Langella is a dedicated researcher of skin care health and products. If you want to know how to turn back the clock for your skin, visit - http://www.totalskinsolution.com.
Why does it happen
Before we talk about choosing a dry skin cream, we need to understand why does our skin get dry in the first place. Broadly, there are two causes of dry skin:
1) Environmental factors - wind, sun and even cold weather cause the skin to become dry
2) Use of soap and harsh cleansers - soaps wash out the natural oils present in our skin, which then makes our skin dry. A similar effect is brought upon our skin by using harsh cleansers. Ideally, a cleanser should only remove extra oil and the dust or pollution that has settled down on the skin. But many cleansers remove all the oils from the skin, making it deficient in oil and hence, dry.
The solution
Some basic skin care combined with a good, natural dry skin cream will work wonders in getting your skin back in shape. A balanced diet is essential for making sure your skin is nourished and gets all the nutrients to keep it healthy. Drink plenty of water - this will keep your body and skin hydrated naturally.
Top it off with a natural skin cream. Now, it's easy to rush off to your neighborhood store and buy the first cream that is packaged attractively and looks good. That, however, is not the best way to do it.
Instead, just do a little research and check the ingredients that a good skin cream should have. Most of the creams available in the stores contain harmful chemicals that do more damage than good for your skin. What is needed is a cream with natural ingredients like Active Manuka Honey. This is a special honey from New Zealand which helps keep the skin hydrated.
Babassu wax is another natural ingredient which softens and soothes the skin. It is made from Babassu oil - sourced from the Babassu palm found in Brazil. It creates a thin, invisible layer on the skin which keeps the dust out and retains the moisture inside.
Do yourself a favor and find out a good natural dry skin cream containing these natural ingredients. You can have a baby-soft smooth skin back in no time!
About the Author
For information on hand lotion to lighten age spots, visit my website today.
Frank Langella is a dedicated researcher of skin care health and products. If you want to know how to turn back the clock for your skin, visit - http://www.totalskinsolution.com.
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